|Web||Searched pages from ucdavis.edu for fu-tong Liu.|
I was not able to read your email. My email program usually shows Chinese characters, but yours was not legible.
May be you can send the text again as an attachment?
Any way, I was very happy to hear from you today and am glad that you have been able to track down many of our classmates. Thank you very much for your effort. I hope we will have a class reunion some time in the near future. My office contact information is listed below.
Professor and Chair
Department of Dermatology
University of California, Davis
School of Medicine
3301 C Street, Suite 1400
Sacramento, CA 95816
Fax: (916) 442-5702
Fu-Tong Liu, M.D. , Ph.D.
Lawrence J. Ellison Ambulatory Care Center
4860 Y St.
Sacramento, CA 95817
||Taipei, , Taiwan|
||National Taiwan University
||University of Miami School of Medicine
||The University of Chicago
||University of California, San Diego
San Diego, California 1990-93
||American Board of Dermatology , 1993|
|American Academy of Dermatology
American Association for the Advancement of Science
American Association of Immunologists
American Chemical Society
American Society for Clinical Investigation
American Society for Investigative Pathology
Society of Investigative Dermatology
Clinical/Research Interests: An allergy and immunology researcher with special interests in skin cancer, Fu-Tong Liu specializes in the study of a family of proteins called galectins that regulate cell activities. As chair of the dermatology department, he is responsible for patient care in UC Davis Medical Center dermatology clinics as well as physician education and research. Liu oversees departmental studies on wound healing and skin aging. He has published more than 100 articles on research related to asthma, immunology and skin cancer.
|Publications: Tinari, N., Kuwabara, I., Huflejt, M., Shen, P.F., Iacobelli, S., Liu, F.-T. 90K/Mac-2BP interacts with galectin-1 and mediates galectin-1-induced cell aggregation. Int. J. Cancer, 91:167-172, 2001.
Liu, F.-T. Role of Galectin-3 in Inflammation. In Lectin and Pathology. Seve, A.P. and Caron, M. eds. Harwood Academic Publishers, pp. 51-65, 2000.
Zuberi, R.I., Apgar,J.R., Chen, S.-S. and Liu, F.-T. A role for IgE in airway secretions: IgE immune complexes are more potent inducers than antigen alone of airway inflammation in a murine model. J. Immunol., 164:2667-2673, 2000.
Matsushita, N., Nishi, N., Seki, M., Matsumoto, R., Kuwabara, I., Liu, F-T., Hata, Y., Nakamura, T., Hirashima, M. Requirement of divalent galactoside-binding activity of ecalectin/galectin-9 for eosinophil chemoattraction. J. Biol. Chem. 275:8355-8360, 2000.
Hsu, D.K., Yang, R.-Y., Pan, Z., Yu, L., Salomon, D.R., Fung-Leung W.-P., and Liu, F.-T. Targeted disruption of the galectin-3 gene results in attenuated peritoneal inflammatory responses. Am. J. Pathol. 156:1073-1083, 2000.
Cortegano,I., del Pozo, V., Cardaba, B., Arrieta, I., Gallardo, S., Rojo, M., Takai, T., Palomino, P., Liu, F.-T., Lahoz, C. Interaction between galectin-3 and FcgRII induces down-regulation of IL-5 gene, implication of the promoter sequence IL-5REIII. Glycobiology, 10:237-242, 2000.
Pugliese,G., Pricci, F., Leto, G., Amadio, L., Iacobini, C., Romeo, G., Lenti, L., Sale, P., Gradini, R., Liu, F.-T., Di Mario, U. The diabetic milieu modulates the glomerular/mesangial expression of galectin-3/age-receptor-3. Diabetes, 49:1249-1257, 2000.
Matarrese, P., Fusco, O., Tinari, N., Natoli, C., Liu, F.-T., Malorni, W., and Iacobelli, S. Overexpression of galectin-3 in human breast carcinoma causes increased cell adhesion and protection from apoptosis. Int. J. Cancer, 85:545-554,2000.
Chen, S.-S., Gong, J., Liu, F.-T., and Mohammed, U. Natural occurring polyphenolic antioxidants modulate IgE-mediated mast cell activation. Immunology, 100:471-480, 2000.
Sano, H., Hsu, D.K., Yu, L., Apgar, J.R. Kuwabara, I., Yamanaka, T., Hirashima, M., Liu, F.-T. Human Galectin-3 is a Novel Chemoattractant for Monocytes and Macrophages, J. Immunol. 165:2156-2164, 2000.
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